William Shaw received his Ph.D. in biochemistry and human physiology from the Medical University of South Carolina and is board certified in clinical chemistry and toxicology. Dr. Shaw has supervised large endocrinology, nutritional biochemistry, toxicology, and immunology departments in positions at the Centers for Disease Control (CDC) and Smith Kline Laboratories in Atlanta, Georgia. As the Director of The Great Plains Laboratory, Inc. in Lenexa, Kansas, he specialises in providing diagnostic tools aiding in the diagnosis and treatment of mental health disorders, mitochondrial disorders, neurological diseases, chronic health issues, and immune diseases.

2017

(1) - Glyphosate, Genetically Modified Organisms( GMOs), Toxicity, and the Impact on the Microbiome of Soil and the Intestine (Sessions 1 and 2)

Glyphosate, an agricultural chemical with a chemical structure similar to phosphate and glycine, has become the leading non-fertilizer agricultural chemical in many countries throughout the world. Several major crops have been genetically modified to withstand glyphosate so that glyphosate can be sprayed on these crops to kill weeds without killing the food crop. In addition, glyphosate is also widely used as a desiccant at harvest for a wide number of GMO crops. The companies producing this herbicide claim it is not harmful to humans because humans do not produce the same enzyme system that is attacked by glyphosate. However, the beneficial bacteria in the soil and the intestinal tracts of humans, mammals, and birds are killed by glyphosate allowing the proliferation of pathogenic bacteria are glyphosate resistant, allowing these pathogens to proliferate in the soil and the intestine. A review of the evidence for the toxicity of glyphosate and its role in carcinogenesis, severe fatal kidney disease, and autism is conducted. Session 1 will cover the background and agricultural uses of glyphosate and session 2 will focus on its disease connections.

(2) - Clostridia bacteria as the cause of neurological and psychiatric disease: critical environmental causes of new epidemics

Clostridia bacteria can be readily detected by mass spectrometry of organic acids in urine. These markers include 3-(3- hydroxyphenyl)-3- hydroxypropionic acid (HPHPA), 4-cresol, 4- hydroxyphenylacetic acid, and 3-indoleacetic acid. Several of these compounds cause illness by inhibition of dopamine-beta- hydroxylase, a critical enzyme that converts dopamine to norepinephrine. The inhibition of this enzyme leads to excess formation of dopamine and decreased formation of epinephrine and norepinephrine in the brain, adrenal glands, and sympathetic nervous system. Excess dopamine is converted into toxic metabolites that can damage neuron structure and function. The treatment of a wide number of diseases is associated with Clostridia overgrowth in the intestinal tract including autism, attention deficit with hyperactivity, depression, tic disorders, obsessive compulsive disorders, eating disorders, schizophrenia, and Parkinson’s disease. The reasons for increased incidence of Clostridia overgrowth are overuse of antibiotics and the wide use of herbicides that kill beneficial bacteria but not Clostridia.