Dr Andrew J Moulden MD PhD

Executive director neurobehavioral research, development and recovery, Rise up and walk institute, Los Angeles, USA. PhD in neuro-electrophysiology, brain imaging and clinical neuropsychology - university of Ottawa, Canada. MD from McMaster university Hamilton Ontario Canada. MA degree in child neorpdevelopment from laurentian university Sudbury Ontario Canada.

Acquired Microvascular ischemic palsies to the cranial nerves: Polio, autism Alzheimer's dementia, and alleged vaccine injuries

Arranging photographic and video records of patients in chronological sequence across time opens a window for assessing brain development and integrity relative to several cranial nerve tracts during pre-morbid mental health states. This analysis allows for a search for putative environmental factors that may have intervened between specific time points wherein the re-constructed cranial nerve examination shows significant deviations in neuromotor integrity across video and photo records.

Cranial nerve partial to complete ischemic palsies create highly specific patterns of neuromotor dissociation. Through video and photographic records of patients, key aspects of the cranial nerve exam in neurology are re-constructed across neurodevelopment and in the pre-morbid state for several mental health, infectious disease, and medical syndromes. A common denominator set of discrete episodes of ischemic-hypoxic microvascular cranial nerve partial palsy signs of acquired brain nerve tract damages are shown for vaccine injuries, Autism, mitochondrial disorders, infantile spasms, mobius syndrome, cerebral palsy from birth hypoxia, brainstem ischemic strokes, sudden infant death syndrome, multiple sclerosis, Alzheimer's, and polio virus induced neuromotor paralysis and respiratory failure. The microvascular nature of these subtle brain damages are most readily appreciated by the emergence of a partial third cranial nerve pupil-sparing palsy and a partial seventh cranial nerve upper motor neuron palsy. It is proposed that pre-post vaccination measures of cranial nerve tract neuromotor functions may provide a window of analysis to detect clinically "silent" damages to the human brain including some cases of repeat vaccination intolerance and adversity in the pre-morbid state.